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These materials are available for nonprofit educational use. This repository is allowed by copyright disclaimer under title 17, Appendix E, section 107 of the United States Copyright Act. Under this statute, allowance is made for “fair use” for purposes such as criticism, comment, news reporting, teaching, scholarship, and research. As defined, fair use specifically applies to this repository.

Medline Title Search (Dupuytren + (Fibrosis or Fibrotic))

Selected Publications

  • Alman BA, Greel DA, Ruby LK, Goldberg MJ, Wolfe HJ. Regulation of proliferation and platelet-derived growth factor expression in palmar fibromatosis (Dupuytren contracture) by mechanical strain. J Orthop Res. 1996/09/01 ed. 1996; Sep;14(5):722–8. (PDF)
  • Berndt A, Kosmehl H, Mandel U, Gabler U, Luo X, Celeda D, et al. TGF beta and bFGF synthesis and localization in Dupuytren’s disease (nodular palmar fibromatosis) relative to cellular activity, myofibroblast phenotype and oncofetal variants of fibronectin. Histochem J. 1995; Dec;27(12):1014–20. (PDF)
  • Calnan J. Fibrosis In Disease. Keloid And Dupuytren’s Contracture. Ann rheum Dis. 1977;36(Supplement):18–22. (PDF)
  • Caroli A, Zanasi S, Marcuzzi A, Guerra D, Cristiani G, Ronchetti IP, et al. Epidemiological and structural findings supporting the fibromatous origin of dorsal knuckle pads. J Hand Surg Br. 1991/08/01 ed. 1991;16(3):258–62. (PDF)
  • Chansky HA, Trumble TE, Conrad EU, Wolff JF, Murray LW, Raskind WH. Evidence for a polyclonal etiology of palmar fibromatosis. J Hand Surg Am. 1999;24(2):339–44. (PDF)
  • Chansky HA, Trumble TE, Conrad EU, Wolff JF, Murray LW, Raskind WH. Evidence for a polyclonal etiology of palmar fibromatosis. J Hand Surg Am. 1999;24(2):339–44. (PDF)
  • Clay RC. Dupuytren’s Contracture: Fibroma of the Palmar Fascia. Ann Surg. 1944;120(2):224–31. (PDF)
  • Diep GK, Agel J, Adams JE. Prevalence of palmar fibromatosis with and without contracture in asymptomatic patients. J Plast Surg Hand Surg. Informa Healthcare; 2015;49(4):247–50. (PDF)
  • Forrester HB, Temple-Smith P, Ham S, de Kretser D, Southwick G, Sprung CN. Genome-Wide Analysis Using Exon Arrays Demonstrates an Important Role for Expression of Extra-Cellular Matrix, Fibrotic Control and Tissue Remodelling Genes in Dupuytren’s Disease. PLoS One. 2013;8(3):e59056. (PDF)
  • Kosmehl H, Berndt A, Katenkamp D, Mandel U, Bohle R, Gabler U, et al. Differential expression of fibronectin splice variants, oncofetal glycosylated fibronectin and laminin isoforms in nodular palmar fibromatosis. Pathol Res Pr. 1995/11/01 ed. 1995;191(11):1105–13. (PDF)
  • Kozma EM, Wisowski G, Kusz D, Olczyk K. The role of decorin and biglycan dermatan sulfate chain(s) in fibrosis-affected fascia. Glycobiology. 2011;21(10):1301–16. (PDF)
  • Kozma EM, Wisowski G, Olczyk K, Kozma EM. Platelet derived growth factor BB is a ligand for dermatan sulfate chain(s) of small matrix proteoglycans from normal and fibrosis affected fascia. Biochimie. 2009;91(11-12):1394–404. (PDF)
  • Krause C, Kloen P, Ten Dijke P. Elevated transforming growth factor ß and mitogen-activated protein kinase pathways mediate fibrotic traits of Dupuytren’s disease fibroblasts. Fibrogenesis Tissue Repair. 2011/06/30 ed. 2011;4(1):14. (PDF)
  • Magro G, Lanteri E, Micali G, Paravizzini G, Travali S, Lanzafame S. Myofibroblasts of palmar fibromatosis co-express transforming growth factor-alpha and epidermal growth factor receptor. J Pathol. 1997;181(2):213–7. (PDF)
  • Meek RMD, Mclellan S, Crossan JF, Crossan SMJF, Rmd M, JF C, et al. Dupuytren’s disease. A model for the mechanism of fibrosis and its modulation by steroids. J Bone Joint Surg Br. 1999;81(4):732–8. (PDF)
  • Meinel AG. Dupuytren contracture – how fibromatosis remodels the palmar subcutaneous tissue and its fibrous environment. Adv Surg Sci. 2013;1(3):11–6. (PDF)
  • Noguchi H, Kephart GM, Colby T V, Gleich GJ. Tissue eosinophilia and eosinophil degranulation in syndromes associated with fibrosis. Am J Pathol. 1992/02/01 ed. 1992;140(2):521–8. (PDF)
  • Van Der Slot AJ, Zuurmond AM, Van Den Bogaerdt AJ, Ulrich MMW, Middelkoop E, Boers W, et al. Increased formation of pyridinoline cross-links due to higher telopeptide lysyl hydroxylase levels is a general fibrotic phenomenon. Matrix Biol. 2004;23(4):251–7. (PDF)
  • Verjee LS, Verhoekx JS, Chan JK, Krausgruber T, Nicolaidou V, Izadi D, et al. Unraveling the signaling pathways promoting fibrosis in Dupuytren’s disease reveals TNF as a therapeutic target. Proc Natl Acad Sci U S A. 2013/02/23 ed. 2013;110(10):E928–37. (PDF)
  • Williams FM, Kalson NS, Fabiane SM, Mann D a., Deehan DJ. Joint Stiffness Is Heritable and Associated with Fibrotic Conditions and Joint Replacement. PLoS One. 2015;10(7):e0133629. (PDF)
  • van Beuge MM, ten Dam E-JPM, Werker PMN, Bank RA. Wnt pathway in Dupuytren’s disease, connecting pro-fibrotic signals. Transl Res. Elsevier Inc.; 2015;166(6):762–71.e3. (PDF)